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GLP-1 receptor signalling promotes β-cell glucose metabolism via mTOR-dependent HIF-1α activation | Scientific Reports
However, the biosimilar pathway for complex peptide medications is accelerating, and several manufacturers have signaled interest in developing competing versions once patents expire
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Because the N-terminus of GLP-1 is critical to its biological function, we engineered the fusion proteins to leave the N-terminus of GLP-1 free
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