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These findings suggest that exendin 9–39 directly interacts with TRPV1, independent of GLP-1R, to inhibit its activation
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These agents, originally developed for glycaemic control in T2DM, have demonstrated promising effects on various cancer-related cellular processes, including the inhibition of tumour cell proliferation, the induction of programmed cell death (apoptosis), and the modulation of angiogenesis—the process by which new blood vessels are formed to supply nutrients to tumour tissues [12]
