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GLP-1R activation has been shown to downregulate proapoptotic proteins such as caspase-3, caspase-9, Bax, and Bcl-2
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Research in decerebrate male rat models further supports the role of the caudal brainstem in GLP-1-mediated cardiovascular responses, where hindbrain-restricted GLP-1R activation was sufficient to suppress food intake while inducing tachycardia, independent of hypothalamic or forebrain input (28)
