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The article says GLP-1 agonists stimulate appetite-suppressing neurons in the hypothalamus and slow gastric emptying, and that during menopause this may counteract the reduced satiety signaling caused by estrogen loss

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At higher doses (5–15 mg/kg), animal studies have shown a reduction in dopaminergic neurons in the substantia nigra and striatum, suggesting potential neurotoxicity that may impair motor and cognitive functions [141,142]